Download PDF by Paula Trahan Rieger: Clinical Handbook for Biotherapy

By Paula Trahan Rieger

ISBN-10: 0763706493

ISBN-13: 9780763706494

This transportable medical guide presents easy accessibility to all significant biologic brokers and their respective nursing administration. person chapters on all significant different types persist with a constant, clinically worthy layout of: assessment of biology, mechanism of motion, regulatory approvals, medical tips, unwanted side effects, administration/dosing, and bibliography.

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198. Ambar BB et al. Treatment of experimental glioma by administration of adenoviral vectors expressing Fas ligand. Hum Gene Therr 1999; 10: 1641-1648. 199. Komata T et al. Caspase-8 gene therapy using the human telomerase reverse transcriptase promoter for malignant glioma cells. Hum Gene Therr 2002; 13: 10151025. 200. Shinoura N et al. Transduction of Apaf-1 or caspase-9 induces apoptosis in A-172 cells that are resistant to p53-mediated apoptosis. Biochem Biophys Res Commun 2000; 272: 667-673.

O'Gorman DM, McKenna SL, McGahon AJ, Cotter TG. Inhibition of PI3-kinase sensitises HL60 human leukaemia cells to both chemotherapeutic drug- and Fasinduced apoptosis by a JNK independent pathway. Leuk Res 2001; 25: 801-811. 157. Haas-Kogan D et al. Protein kinase B (PKB/Akt) activity is elevated in glioblastoma cells due to mutation of the tumor suppressor PTEN/MMAC. Curr Bioll 1998; 8: 11951198. 158. Karin M, Ben-Neriah Y. Phosphorylation meets ubiquitination: the control of NF[kappa]B activity.

Morphological changes and DNA fragmentation, typical of apoptotic cell death, were detected in MCF-7 cells only subsequent to the secondary [Ca2+]in rise, after about 48 hours of TG exposure [93]. These observations suggested that the secondary rise in [Ca2+]in to micromolar levels is not prostate-specific, but rather reflects a general feature of Ca2+ signaling required for endonucleases activation during TG-induced cancer cells apoptosis. In contrast, an activation of Ca2+/Mg2+-dependent endonuclease was detected as an early event in MCF-7 cells apoptosis induced by a widely used anticancer drug, etoposide (VP-16) [94].

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Clinical Handbook for Biotherapy by Paula Trahan Rieger

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